TRIM37 mitigates adriamycin-induced nephropathy by promoting PTEN ubiquitination and degradation to suppress mitochondrial apoptosis and podocyte injury
Human & Experimental Toxicology
Published online on March 14, 2026
Abstract
Human & Experimental Toxicology, Volume 45, January-December 2026.
IntroductionPodocyte loss or functional impairment is a key event in the pathophysiology of nephrotic syndrome (NS). NS can further progress to chronic kidney disease (CKD) and/or end-stage renal disease (ESRD). This study aimed to explore the potential ...
IntroductionPodocyte loss or functional impairment is a key event in the pathophysiology of nephrotic syndrome (NS). NS can further progress to chronic kidney disease (CKD) and/or end-stage renal disease (ESRD). This study aimed to explore the potential ...